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Protein kinase CK2 in hematologic malignancies: reliance on a pivotal cell survival regulator by oncogenic signaling pathways

Author
PIAZZA, F1 3 ; MANNI, S1 3 ; RUZZENE, M2 3 ; PINNA, L. A2 3 ; GURRIERI, C1 3 ; SEMENZATO, G1 3
[1] Department of Clinical and Experimental Medicine, Hematology and Clinical Immunology Branch, University of Padua School of Medicine, Padua, Italy
[2] Department of Biological Chemistry, University of Padua, Padua, Italy
[3] Venetian Institute of Molecular Medicine, Hematological Malignancies Unit, Padua, Italy
Source

Leukemia. 2012, Vol 26, Num 6, pp 1174-1179, 6 p ; ref : 60 ref

CODEN
LEUKED
ISSN
0887-6924
Scientific domain
Medical oncology; Hematology
Publisher
Nature Publishing Group, Avenel, NJ
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
blood tumors hematopoiesis kinase inhibitors protein kinase CK2
Keyword (fr)
Carcinogenèse Hématologie Hématopoïèse Hémopathie maligne Inhibiteur Kinase Régulateur Sang Transduction signal Tumeur Casein kinase II Survie cellulaire Voie de signalisation Cancer Enzyme Transferases
Keyword (en)
Carcinogenesis Hematology Hematopoiesis Malignant hemopathy Inhibitor Kinase Regulator Blood Signal transduction Tumor Casein kinase II Cell survival Signaling pathway Cancer Enzyme Transferases
Keyword (es)
Carcinogénesis Hematología Hematopoyesis Hemopatía maligna Inhibidor Kinase Regulador Sangre Transducción señal Tumor Supervivencia celular Vía de señalización Cáncer Enzima Transferases
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B19 Hematologic and hematopoietic diseases

Discipline
Blood diseases
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
25986184

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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