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Control of cell cycle progression in human natural killer cells through redox regulation of expression and phosphorylation of retinoblastoma gene product protein

Author
YAMAUCHI, A1 ; BLOOM, E. T1
[1] Laboratory of Cellular Immunology (HFM-518), Division of Cellular and Gene Therapies, Center for Biologics Evaluation and Research, Food and Drug Administration, Bethesda, MD, United States
Source

Blood. 1997, Vol 89, Num 11, pp 4092-4099 ; ref : 36 ref

ISSN
0006-4971
Scientific domain
Medical oncology; Hematology
Publisher
The Americain Society of Hematology, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Cellule NK Cycle cellulaire Expression génique Gène rétinoblastome Gène suppresseur tumeur Homme In vitro Lignée cellulaire établie Lymphocyte Multiplication cellulaire Oxydoréduction Phosphorylation Protein kinase Régulation Transduction signal Lignée NK3 3 Enzyme Transferases
Keyword (en)
Natural killer cell Cell cycle Gene expression Retinoblastoma gene Tumor suppressor gene Human In vitro Established cell line Lymphocyte Cell proliferation Oxidation reduction Phosphorylation Protein kinase Regulation(control) Signal transduction Enzyme Transferases
Keyword (es)
Célula NK Ciclo celular Expresión genética Gen retinoblastoma Gen supresor tumor Hombre In vitro Línea celular establecida Linfocito Multiplicación celular Oxidación reducción Fosforilación Protein kinase Regulación Transducción señal Enzima Transferases
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H02 Cell cycle, cell proliferation

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
2680898

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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