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γ-Interferon-induced resistance to 1,25-(OH)2 D3in human monocytes and macrophages : A mechanism for the hypercalcemia of various granulomatoses

Author
DUSSO, A. S1 ; KAMIMURA, S1 ; GALLIENI, M1 ; ZHONG, M1 ; NEGREA, L1 ; SHAPIRO, S2 ; SLATOPOLSKY, E1
[1] The Division of Nephrology, Department of Internal Medicine, Washington University Medical Center, St. Louis, Missouri 63110, United States
[2] The Division of Respiratory and Critical Care, Department of Internal Medicine, Washington University Medical Center, St. Louis, Missouri 63110, United States
Source

The Journal of clinical endocrinology and metabolism. 1997, Vol 82, Num 7, pp 2222-2232 ; ref : 57 ref

CODEN
JCEMAZ
ISSN
0021-972X
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
Endocrine Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Antagonisme Cholécalciférol Elément minéral Granulomatose Homme Hypercalcémie Interféron gamma Macrophage Monocyte Pathogénie Plasma sanguin Propriété biologique Résistance Cytokine Trouble métabolisme Trouble équilibre hydroélectrolytique
Keyword (en)
Antagonism Colecalciferol Inorganic element Granulomatosis Human Hypercalcemia Gamma interferon Macrophage Monocyte Pathogenesis Blood plasma Biological properties Resistance Cytokine Metabolic disorder Hydroelectrolytic balance disorder
Keyword (es)
Antagonismo Colecalciferol Elemento inorgánico Granulomatosis Hombre Hipercalcemia Interferón gamma Macrófago Monocito Patogenia Plasma sanguíneo Propiedad biológica Resistencia Citoquina Trastorno metabolismo Trastorno equilibrio hidroelectrolítico
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B07 Sarcoidosis. Granulomatous diseases of unproved etiology. Connective tissue diseases. Elastic tissue diseases. Vasculitis

Discipline
Sarcoidosis. Granulomatosis of undetermined origin. Connective tissue diseases. Vasculitides
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
2739012

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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