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Teplizumab treatment may improve C-peptide responses in participants with type 1 diabetes after the new-onset period: a randomised controlled trial

Author
HEROLD, K. C1 ; GITELMAN, S. E2 ; MICHELS, A. W4 ; DZIURA, J6 ; BLUESTONE, J. A2 ; WILLI, S. M3 ; GOTTLIEB, P. A4 ; WALDRON-LYNCH, F1 ; DEVINE, L1 ; SHERR, J5 ; ROSENTHAL, S. M2 ; ADI, S2 ; JALALUDIN, M. Y3
[1] Department of Immunobiology and Internal Medicine, Yale University, 300 George St, 353E, New Haven, CT 06511, United States
[2] Department of Pediatrics and Internal Medicine, University of California at San Francisco, San Francisco, CA, United States
[3] Department of Pediatrics, Children's Hospital of Philadelphia, Philadelphia, PA, United States
[4] Department of Internal Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO, United States
[5] Department of Pediatrics, Yale University, New Haven, CT, United States
[6] Department of Emergency Medicine, Yale University, New Haven, CT, United States
Source

Diabetologia (Berlin). 2013, Vol 56, Num 2, pp 391-400, 10 p ; ref : 31 ref

ISSN
0012-186X
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
Springer, Heidelberg
Publication country
Germany
Document type
Article
Language
English
Author keyword
Autoimmunity Immune therapy Type 1 diabetes
Keyword (fr)
Autoimmunité Diabète de type 1 Peptide C Traitement Endocrinopathie Immunopathologie Maladie autoimmune
Keyword (en)
Autoimmunity Type 1 diabetes C-Peptide Treatment Endocrinopathy Immunopathology Autoimmune disease
Keyword (es)
Autoinmunidad Diabetes de tipo 1 Péptido C Tratamiento Endocrinopatía Inmunopatología Enfermedad autoinmune
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01A Etiopathogenesis. Screening. Investigations. Target tissue resistance

Discipline
Endocrinopathies
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
27531367

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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