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The participation of the Fas-mediated cytotoxic pathway by natural killer cells is tumor-cell-dependent

Author
MORI, S1 ; JEWETT, A1 ; MURAKAMI-MORI, K2 ; CAVALCANTI, M1 ; BONAVIDA, B1
[1] Department of Microbiology and Immunobiology, UCLA School of Medicine, University of California, 10833 Le Conte Ave., A2084 CHS, Los Angeles, CA 900951747, United States
[2] Institute of Molecular Medicine, Huntington Memorial Hospital, Pasadena, CA 91105, United States
Source

Cancer immunology and immunotherapy. 1997, Vol 44, Num 5, pp 282-290 ; ref : 30 ref

CODEN
CIIMDN
ISSN
0340-7004
Scientific domain
Biochemistry, molecular biology, biophysics; Medical oncology; Immunology, immunopathology, allergology
Publisher
Springer, Berlin
Publication country
Germany
Document type
Article
Language
English
Keyword (fr)
Anticancéreux Cellule NK Cellule tumorale Cytokine Cytotoxicité Granzyme A Granzyme B Homme Immunité cellulaire In vitro Mort cellulaire Mécanisme Perforine Protéine fas Enzyme Hydrolases Peptidases Serine endopeptidases
Keyword (en)
Antineoplastic agent Natural killer cell Tumor cell Cytokine Cytotoxicity Granzyme A Granzyme B Human Cellular immunity In vitro Cell death Mechanism Enzyme Hydrolases Peptidases Serine endopeptidases
Keyword (es)
Anticanceroso Célula NK Célula tumoral Citoquina Citotoxicidad Granzyme A Granzyme B Hombre Inmunidad celular In vitro Muerte celular Mecanismo Enzima Hydrolases Peptidases Serine endopeptidases
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C05 Analysis of the immune response. Humoral and cellular immunity / 002A06C05A Organs and cells involved in the immune response

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors / 002B04F Host-tumor relations. Immunology. Biological markers

Discipline
Fundamental immunology Tumours
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
2753347

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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