Abnormal splicing of the leptin receptor in diabetic mice
- Author
- LEE, G.-H1 ; PROENCA, R1 ; MONTEZ, J. M; CARROLL, K. M; DARVISHZADEH, J. G; LEE, J. I; FRIEDMAN, J. M1
[1] Rockefeller univ., Howard Hughes medical inst., New York NY 10021, United States - Source
Nature (London). 1996, Vol 379, Num 6566, pp 632-635 ; ref : 27 ref
- CODEN
- NATUAS
- ISSN
- 0028-0836
- Scientific domain
- Multidisciplinary
- Publisher
- Nature Publishing, London
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Keyword (fr)
- Diabète Epissage Etat nutritionnel Modèle animal Mutation Obésité Produit gène Récepteur biologique Souris Transduction signal Gène db Leptine Endocrinopathie Mammalia Rodentia Trouble nutrition Vertebrata
- Keyword (en)
- Diabetes mellitus Splicing Nutritional status Animal model Mutation Obesity Gene product Biological receptor Mouse Signal transduction Leptin Endocrinopathy Mammalia Rodentia Nutrition disorder Vertebrata
- Keyword (es)
- Diabetes Empalme Estado nutricional Modelo animal Mutación Obesidad Producto gene Receptor biológico Ratón Transducción señal Endocrinopatía Mammalia Rodentia Trastorno nutricíon Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01B Associated diseases and complications
- Discipline
- Endocrinopathies
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 2981341
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/379632a0 
https://dx.doi.org/10.1038/379632a0
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