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Potentiation of enediyne-induced apoptosis and differentiation by Bcl-2

Author
CORTAZZO, M1 ; SCHOR, N. F
[1] Department of Neuroscience, University of Pittsburgh, Pittsburgh, Pennsylvania 15213, United States
Source

Cancer research (Baltimore). 1996, Vol 56, Num 6, pp 1199-1203 ; ref : 18 ref

CODEN
CNREA8
ISSN
0008-5472
Scientific domain
Medical oncology; Pharmacology drugs
Publisher
American Association for Cancer Research, Philadelphia, PA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Animal Anticancéreux Apoptose Cellule tumorale Composé acétylénique Composé diynique Cytotoxicité Glutathion In vitro Intracellulaire Lignée cellulaire établie Mort cellulaire Mécanisme action Potentiel oxydoréduction Rat Sensibilité Surexpression génique Thiol Zinostatine Gène Bcl-2 Lignée PC12 Mammalia Rodentia Vertebrata
Keyword (en)
Animal Antineoplastic agent Apoptosis Tumor cell Acetylenic compound Diynic compound Cytotoxicity Glutathione In vitro Intracellular Established cell line Cell death Mechanism of action Redox potential Rat Sensitivity Gene overexpression Thiol Mammalia Rodentia Vertebrata
Keyword (es)
Animal Anticanceroso Apoptosis Célula tumoral Compuesto acetilénico Compuesto diínico Citotoxicidad Glutatión In vitro Intracelular Línea celular establecida Muerte celular Mecanismo acción Potencial redox Rata Sensibilidad Sobreexpressión genética Tiol Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02R Antineoplastic agents / 002B02R01 General aspects

Discipline
Pharmacological treatments
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
3029626

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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