Targeted disruption of the neurotrophin-3 gene with lacZ induces loss of trkC-positive neurons in sensory ganglia but not in spinal cords
- Author
- TOJO, H1 ; KAISHO, Y1 ; AIZAWA, S; SHIHO, O; NAKATA, M1 ; MATSUOKA, K1 ; KITAGAWA, M; ABE, T; TAKAMI, K; YAMAMOTO, M; SHINO, A; IGARASHI, K1
[1] Takeda Chemical Industries, Ltd, Discovery Research Laboratories II, Yodogawa-ku, Osaka 532, Japan - Source
Brain research. 1995, Vol 669, Num 2, pp 163-175 ; ref : 62 ref
- CODEN
- BRREAP
- ISSN
- 0006-8993
- Scientific domain
- Neurology; Physiology, morphology
- Publisher
- Elsevier, Amsterdam
/
Elsevier, London
/
Elsevier, New York, NY
- Publication country
- Netherlands
- Document type
- Article
- Language
- English
- Keyword (fr)
- Développement Expression génique Moelle épinière Mutation Neurone sensoriel Souris Gène lacZ Neurotrophine 3 Récepteur trkC Mammalia Rodentia Système nerveux central Vertebrata
- Keyword (en)
- Development Gene expression Spinal cord Mutation Sensory neuron Mouse Neurotrophin 3 Mammalia Rodentia Central nervous system Vertebrata
- Keyword (es)
- Desarrollo Expresión genética Médula espinal Mutación Neurona sensorial Ratón Mammalia Rodentia Sistema nervioso central Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A25 Vertebrates: nervous system and sense organs / 002A25D Central nervous system / 002A25D02 Biochemistry and metabolism
- Discipline
- Vertebrates : nervous system and sense organs
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 3361203
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1016/0006-8993(94)01219-8 
https://dx.doi.org/10.1016/0006-8993(94)01219-8
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