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Blocking of tumor promoter-induced AP-1 activity inhibits induced transformation in JB6 mouse epidermal cells

Author
ZIGANG DONG1 ; BIRRER, M. J; WATTS, R. G; MATRISIAN, L. M; COLBURN, N. H
[1] PRI/DynCorp., biological carcinogenesis development program, Frederick MD 21702-1201, United States
Source

Proceedings of the National Academy of Sciences of the United States of America. 1994, Vol 91, Num 2, pp 609-613 ; ref : 34 ref

CODEN
PNASA6
ISSN
0027-8424
Scientific domain
Multidisciplinary
Publisher
National Academy of Sciences of the United States of America, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Carcinogenèse Epiderme Facteur croissance épiderme Gène onc cellulaire Induction Lignée cellulaire Mécanisme Phorbol ester Promoteur tumeur Souris Transformation cellulaire Facteur transcription AP1 Gène jun Lignée JB6 Mammalia Rodentia Vertebrata
Keyword (en)
Carcinogenesis Epidermis Epidermal growth factor C-Onc gene Induction Cell line Mechanism Tumor promotor Mouse Cell transformation Transcription factor AP1 Mammalia Rodentia Vertebrata
Keyword (es)
Carcinogénesis Epidermis Factor crecimiento epidermi Gen onc celular Inducción Línea celular Mecanismo Promotor tumor Ratón Transformación celular Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
3923358

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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