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High affinity VEGF binding and developmental expression suggest Flk-1 as a major regulator of vasculogenesis and angiogenesis

Author
MILLAUER, B1 ; WIZIGMANN-VOOS, S; SCHNÜRCH, H; MARTINEZ, R; MØLLER, N. P. H1 ; RISAU, W; ULLRICH, A1
[1] Max Planck inst. biochemistry, dep. molecular biology, 8033 Martinsried, Germany
Source

Cell (Cambridge). 1993, Vol 72, Num 6, pp 835-846 ; ref : 1 p. 3/4

CODEN
CELLB5
ISSN
0092-8674
Scientific domain
Biochemistry, molecular biology, biophysics; Cell biology, histology
Publisher
Cell Press, Cambridge, MA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Angiogenèse Cellule endothéliale Coopérativité moléculaire Développement embryonnaire Expression génique Facteur croissance Produit gène Protein-tyrosine kinase Récepteur membranaire Souris Vascularisation Facteur croissance endothélium vasculaire Gène flk1 Enzyme Mammalia Rodentia Vertebrata
Keyword (en)
Angiogenesis Endothelial cell Molecular cooperativity Embryonic development Gene expression Growth factor Gene product Protein-tyrosine kinase Membrane receptor Mouse Vascularization Enzyme Mammalia Rodentia Vertebrata
Keyword (es)
Angiogénesis Célula endotelial Cooperatividad molecular Desarrollo embrionario Expresión genética Factor crecimiento Producto gene Protein-tyrosine kinase Receptor membrana Ratón Vascularización Enzima Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H03 Cell differentiation, maturation, development, hematopoiesis

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
4658593

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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