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Evidence that the T cell repertoire of normal rats contains cells with the potential to cause diabetes. Characterization of the CD4+ T cell subset that inhibits this autoimmune potential

Author
FOWELL, D; MASON, D
MRC cellular immunology unit, Sir William Dunn school pathology, Oxford Oxon OX1 3RE, United Kingdom
Source

The Journal of experimental medicine. 1993, Vol 177, Num 3, pp 627-636 ; ref : 70 ref

CODEN
JEMEAV
ISSN
0022-1007
Scientific domain
Biochemistry, molecular biology, biophysics; Immunology, immunopathology, allergology; General medicine general surgery
Publisher
Rockefeller University Press, New York, NY
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Autoimmunité Diabète Immunorégulation Irradiation gamma Lymphocyte T Modèle animal Pathogénie Pathologie expérimentale Rat Sous population cellulaire Thymectomie Antigène CD4 Antigène CD8 Répertoire immunologique Endocrinopathie Immunopathologie Mammalia Rodentia Vertebrata
Keyword (en)
Autoimmunity Diabetes mellitus Immune regulation Gamma irradiation T-Lymphocyte Animal model Pathogenesis Experimental disease Rat Cell subpopulation Thymectomy Endocrinopathy Immunopathology Mammalia Rodentia Vertebrata
Keyword (es)
Autoinmunidad Diabetes Inmunoregulación Irradiación gama Linfocito T Modelo animal Patogenia Patología experimental Rata Subpoblación celular Timectomía Endocrinopatía Inmunopatología Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01A Etiopathogenesis. Screening. Investigations. Target tissue resistance

Discipline
Endocrinopathies
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
4697971

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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