Neutrophils contribute to hepatic ischemia-reperfusion injury by a CD18-independent mechanism
- Author
- LANGDALE, L. A; FLAHERTY, L. C; LIGGITT, H. D; HARLAN, J. M; RICE, C. L; WINN, R. K
Univ. Washington, dep. surgery, Seattle WA, United States - Source
Journal of leukocyte biology. 1993, Vol 53, Num 5, pp 511-517 ; ref : 20 ref
- Publisher
- Federation of American Societies for Experimental Biology, Bethesda, MD
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Adhérence Anticorps monoclonal Cellule endothéliale Foie pathologie Inflammation Ischémie Lapin Neutrophile Neutropénie Pathogénie Récepteur membranaire Vinblastine Antigène CD18 Appareil circulatoire pathologie Hémopathie Lagomorpha Mammalia Vaisseau sanguin pathologie Vertebrata
- Keyword (en)
- Adhesion Monoclonal antibody Endothelial cell Hepatic disease Inflammation Ischemia Rabbit Neutrophil Neutropenia Pathogenesis Membrane receptor Cardiovascular disease Hemopathy Lagomorpha Mammalia Vascular disease Vertebrata
- Keyword (es)
- Adherencia Anticuerpo monoclonal Célula endotelial Hígado patología Inflamación Isquemia Conejo Neutrófilo Neutropenia Patogenia Receptor membrana Aparato circulatorio patología Hemopatía Lagomorpha Mammalia Vaso sanguíneo patología Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04J Inflammation
- Discipline
- Molecular and cell biology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 4768600
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1002/jlb.53.5.511 
https://dx.doi.org/10.1002/jlb.53.5.511
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