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Enforced BCL2 expression in B-lymphoid cells prolongs antibody responses and elicits autoimmune disease

Author
STRASSER, A; WHITTINGHAM, S; VAUX, D. L; BATH, M. L; ADAMS, J. M; CORY, S; HARRIS, A. W
Royal Melbourne hosp., Walter Eliza Hall inst. medical res., Victoria 3050, Australia
Source

Proceedings of the National Academy of Sciences of the United States of America. 1991, Vol 88, Num 19, pp 8661-8665 ; ref : 33 ref

CODEN
PNASA6
ISSN
0027-8424
Scientific domain
Multidisciplinary
Publisher
National Academy of Sciences of the United States of America, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Animal transgénique Autoimmunité Cellule lymphoïde Cellule sécrétrice Expression génique Gène Immunité humorale Immunoglobuline Immunomodulation Lymphocyte B Souris Survie Gène BCL2 Mammalia Rodentia Réponse immune Vertebrata
Keyword (en)
Transgenic animal Autoimmunity Lymphoid cell Secretory cell Gene expression Gene Humoral immunity Immunoglobulins Immunomodulation B-Lymphocyte Mouse Survival Mammalia Rodentia Immune response Vertebrata
Keyword (es)
Animal transgénico Autoinmunidad Célula linfoide Célula secretoria Expresión genética Gen Inmunidad humoral Inmunoglobulina Inmunomodulación Linfocito B Ratón Sobrevivencia Mammalia Rodentia Respuesta inmune Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C08 Modulation of the immune response (stimulation, suppression)

Discipline
Fundamental immunology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
5011180

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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