Death of cultured hippocampal pyramidal neurons induced by pathological activation of N-methyl-D-aspartate receptors is reduced by monosialogangliosides
- Author
- SKAPER, S. D; LEON, A; FACCI, L
Fidia Research Laboratories, Abanno Terme, Italy - Source
The Journal of pharmacology and experimental therapeutics. 1991, Vol 259, Num 1, pp 452-457 ; ref : 1 p
- CODEN
- JPETAB
- ISSN
- 0022-3565
- Scientific domain
- Pharmacology drugs
- Publisher
- American Society for Pharmacology and Experimental Therapeutics, Bethesda, MD
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Aminoacide excitateur Animal Encéphale Etude comparative Ganglioside Glutamate Hippocampe In vitro Ischémie Mort cellulaire Neurone pyramidal Neurotoxine Prévention Rat Récepteur NMDA Récepteur glutamate Système nerveux pathologie Mammalia Rodentia Vertebrata
- Keyword (en)
- Excitatory aminoacid Animal Brain (vertebrata) Comparative study Ganglioside Glutamate Hippocampus In vitro Ischemia Cell death Pyramidal neuron Neurotoxin Prevention Rat NMDA receptor Glutamate receptor Nervous system diseases Mammalia Rodentia Vertebrata
- Keyword (es)
- Aminoácido excitador Animal Encéfalo Estudio comparativo Gangliósido Glutamato Hipocampo In vitro Isquemia Muerte celular Neurona piramidal Neurotoxina Prevención Rata Receptor NMDA Receptor glutámato Sistema nervioso patología Mammalia Rodentia Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02B Neuropharmacology / 002B02B11 Miscellaneous
- Discipline
- Pharmacological treatments
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 5045432
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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