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A synthetic peptide homologous to retrovial transmembrane envelope proteins depresses protein kinase C mediated lymphocyte proliferation and directly inactivated protein kinase C : a potential mechanism for immunosuppression

Author
KADOTA, J; CIANCIOLO, G. J; SNYDERMAN, R
Nagasaki univ. school medicine, second dep. internal medicine, Nagasaki 852, Japan
Source

Microbiology and immunology. 1991, Vol 35, Num 6, pp 443-459 ; ref : 51 ref

CODEN
MIIMDV
ISSN
0385-5600
Scientific domain
Biochemistry, molecular biology, biophysics; Immunology, immunopathology, allergology; Microbiology, infectious diseases
Publisher
Center for Academic Publications Japan, Tokyo
Publication country
Japan
Document type
Article
Language
English
Keyword (fr)
Enveloppe virus Homme Immunodépression Lymphocyte Multiplication cellulaire Mécanisme Phorbol ester Produit synthétique Protein kinase C Protéine transmembranaire Protéine Retroviridae Régulation Virose Enzyme Infection Virus
Keyword (en)
Virus envelope Human Immunosuppression Lymphocyte Cell proliferation Mechanism Synthetic product Protein kinase C Transmembrane protein Proteins Retroviridae Regulation(control) Viral disease Enzyme Infection Virus
Keyword (es)
Cubierta virus Hombre Inmunodepresión Linfocito Multiplicación celular Mecanismo Producto sintético Protein kinase C Proteína transmembranar Proteina Retroviridae Regulación Virosis Enzima Infección Virus
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C08 Modulation of the immune response (stimulation, suppression)

Discipline
Fundamental immunology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
5100661

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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