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Negative and positive promoter elements contribute to tissue specificity of apolipoprotein B expression

Author
CARLSSON, P1 ; BJURSELL, G
[1] Univ. Göteborg, dep. medical biochemistry, Göteborg 400 33, Sweden
Source

Gene (Amsterdam). 1989, Vol 77, Num 1, pp 113-121 ; ref : 28 ref

CODEN
GENED6
ISSN
0378-1119
Scientific domain
Cell biology, histology; Biotechnology; Genetics
Publisher
Elsevier, Amsterdam / Elsevier, Lausanne / Elsevier, New York, NY
Publication country
Netherlands
Document type
Article
Language
English
Keyword (fr)
Apolipoprotéine B Clonage moléculaire DNA Délétion Expression génique Foie Homme Intestin Lignée HeLa Lignée cellulaire Lipoprotéine Promoteur transcription Régulation Spécificité tissu Séquence nucléotide Séquence régulatrice Gène apo B Lignée CaCo-2 Lignée Hep G2
Keyword (en)
Apolipoprotein B Molecular cloning DNA Deletion Gene expression Liver Human Gut Hela cell line Cell line Lipoprotein Transcription promoter Regulation(control) Tissue specificity Nucleotide sequence Regulatory sequence
Keyword (es)
Apolipoproteína B Clonación molecular DNA Deleción Expresión genética Hígado Hombre Intestino Célula HeLa Línea celular Lipoproteina Promotor transcripción Regulación Especificidad tejido Secuencia nucleótido Secuencia reguladora
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04C Molecular genetics / 002A04C05 Transcription. Transcription factor. Splicing. Rna processing

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
6671852

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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