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A role of HLA-DQ molecules of stimulator-adherent cells in the regulation of human autologous mixed lymphocyte reaction

Author
NIEDA, M1 ; JUJI, T; IMAO, S; MINAMI, M
[1] Tokyo univ., hosp., blood transfusion serv., Bunkyo-ku Tokyo 113, Japan
Source

The Journal of immunology (1950). 1988, Vol 141, Num 9, pp 2975-2979 ; ref : 27 ref

CODEN
JOIMA3
ISSN
0022-1767
Scientific domain
Biochemistry, molecular biology, biophysics; Immunology, immunopathology, allergology
Publisher
American Association of Immunologists, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Antigène histocompatibilité classe II Cellule suppressive Homme Immunité cellulaire Induction Lymphocyte T Réaction lymphocytaire mixte Régulation Sous population cellulaire Système autologue Système histocompatibilité majeur Antigène HLA-DQ
Keyword (en)
Class II histocompatibility antigen Suppressive cell Human Cellular immunity Induction T-Lymphocyte Mixed lymphocyte reaction Regulation(control) Cell subpopulation Autologous system Major histocompatibility system
Keyword (es)
Antígeno histocompatibilidad clase II Célula supresora Hombre Inmunidad celular Inducción Linfocito T Reacción linfocitaria mixta Regulación Subpoblación celular Sistema autólogo Sistema histocompatibilidad mayor
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C11 Immunological reactions in vitro / 002A06C11A Lymphoblastic transformation (stimulation by antigen, mitogen, mixed lymphocyte reactions)

Discipline
Fundamental immunology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
7206317

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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