Cell type specific involvement of death receptor and mitochondrial pathways in drug-induced apoptosis
- Author
- FULDA, Simone1 ; MEYER, Eric1 ; FRIESEN, Claudia1 ; SUSIN, Santos A2 ; KROEMER, Guido2 ; DEBATIN, Klaus-Michael1
[1] University Children's Hospital, Prittwitzstr. 43, 89075 Ulm, Germany
[2] Centre Nationale de la Recherche Scientifique, Unité Mixte de Recherche 1599, Institut Gustave Roussy, 39 rue Camille Desmoulins, 94805 Villejuif, France - Source
Oncogene (Basingstoke). 2001, Vol 20, Num 9, pp 1063-1075 ; ref : 1 p.1/4
- ISSN
- 0950-9232
- Scientific domain
- Cell biology, histology; Medical oncology; Genetics
- Publisher
- Nature Publishing, Basingstoke
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Keyword (fr)
- Anticancéreux Apoptose Carcinogenèse Cellule tumorale Homme Induction Lignée cellulaire Mitochondrie Récepteur biologique Transduction signal Caspase Protéine Fas Cysteine endopeptidases Enzyme Hydrolases Peptidases
- Keyword (en)
- Antineoplastic agent Apoptosis Carcinogenesis Tumor cell Human Induction Cell line Mitochondria Biological receptor Signal transduction Cysteine endopeptidases Enzyme Hydrolases Peptidases
- Keyword (es)
- Anticanceroso Apoptosis Carcinogénesis Célula tumoral Hombre Inducción Línea celular Mitocondria Receptor biológico Transducción señal Cysteine endopeptidases Enzima Hydrolases Peptidases
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death
- Discipline
- Molecular and cell biology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 994876
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/sj.onc.1204141 
https://dx.doi.org/10.1038/sj.onc.1204141
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