Minimal residual disease in chronic myeloid leukaemiaCROSS, N. C. P.Hematology and cell therapy (Print). 1998, Vol 40, Num 5, pp 224-228, issn 1269-3286Conference Paper

Partial aldolase B gene deletions in hereditary fructose intoleranceCROSS, N. C. P; COX, T. M.American journal of human genetics. 1990, Vol 47, Num 1, pp 101-106, issn 0002-9297, 6 p.Article

Assessing residual leukaemiaCROSS, N. C. P.Baillière's clinical haematology. 1997, Vol 10, Num 2, pp 389-403, issn 0950-3536Article

A novel arrangement of sequence elements surrounding the rDNA promoter and its spacer duplications in Tsetse speciesCROSS, N. C. P; DOVER, G. A.Journal of molecular biology. 1987, Vol 195, Num 1, pp 63-74, issn 0022-2836Article

Tyrosine kinase fusion genes in chronic myeloproliferative diseases : Spotlight on signal transduction inhibitors: Basic aspects and therapeutics implicationsCROSS, N. C. P; REITER, A.Leukemia. 2002, Vol 16, Num 7, pp 1207-1212, issn 0887-6924Article

Tsetse fly rDNA: an analysis of structure and sequenceCROSS, N. C. P; DOVER, G. A.Nucleic acids research. 1987, Vol 15, Num 1, pp 15-30, issn 0305-1048Article

Catalytic deficiency of human aldolase B in hereditary fructose intolerance caused by a common missense mutationCROSS, N. C. P; TOLAN, D. R; COX, T. M et al.Cell (Cambridge). 1988, Vol 53, Num 6, pp 881-885, issn 0092-8674Article

Rarity of microsatellite alterations in acute myeloid leukaemiaSILL, H; GOLDMAN, J. M; CROSS, N. C. P et al.British journal of cancer. 1996, Vol 74, Num 2, pp 255-257, issn 0007-0920Article

Dominant-negative mutations of the Wilms' tumour predisposing gene (WT1) are infrequent in CML blast crisis and de novo acute leukaemiaCARAPETI, M; GOLDMAN, J. M; CROSS, N. C. P et al.European journal of haematology. 1997, Vol 58, Num 5, pp 346-349, issn 0902-4441Article

Homozygous deletions of the p16 tumor-suppressor gene are associated with lymphoid transformation of chronic myeloid leukemiaSILL, H; GOLDMAN, J. M; CROSS, N. C. P et al.Blood. 1995, Vol 85, Num 8, pp 2013-2016, issn 0006-4971Article

A new aldolase B variant, N334K, is a common cause of hereditary fructose intolerance in YugoslaviaCROSS, N. C. P; STOJANOV, L. M; COX, T. M et al.Nucleic acids research. 1990, Vol 18, Num 7, issn 0305-1048, p. 1925Article

Characterization of genomic BCR-ABL breakpoints in chronic myeloid leukaemia by PCRZHANG, J. G; GOLDMAN, J. M; CROSS, N. C. P et al.British journal of haematology. 1995, Vol 90, Num 1, pp 138-146, issn 0007-1048Article

A comparison of the sensitivity of blood and bone marrow for the detection of minimal residual disease in chronic myeloid leukaemiaFENG LIN; GOLDMAN, J. M; CROSS, N. C. P et al.British journal of haematology. 1994, Vol 86, Num 3, pp 683-685, issn 0007-1048Article

Imatinib sensitivity as a consequence of a CSF1R-Y571D mutation and CSF1/CSF1R signaling abnormalities in the cell line GDM1CHASE, A; SCHULTHEIS, B; KREIL, S et al.Leukemia. 2009, Vol 23, Num 2, pp 358-364, issn 0887-6924, 7 p.Article

P2X7 polymorphism and chronic lymphocytic leukaemia: lack of correlation with incidence, survival and abnormalities of chromosome 12ZHANG, L. Y; IBBOTSON, R. E; ORCHARD, J. A et al.Leukemia. 2003, Vol 17, Num 11, pp 2097-2100, issn 0887-6924, 4 p.Article

Comparison of single-dose and escalating-dose regimens of donor lymphocyte infusion for relapse after allografting for chronic myeloid leukemiaDAZZI, F; SZYDLO, R. M; GOLDMAN, J. M et al.Blood. 2000, Vol 95, Num 1, pp 67-71, issn 0006-4971Article

Fusion of H4/D10S170 to the platelet-derived growth factor receptor β in BCR-ABL-negative myeloproliferative disorders with a t(5;10)(q33;q21)KULKARNI, S; HEATH, C; PARKER, S et al.Cancer research (Baltimore). 2000, Vol 60, Num 13, pp 3592-3598, issn 0008-5472Article

Frequent deletion of hSNF5/INI1, a component of the SWI/SNF complex, in chronic myeloid leukemiaGRAND, F; KULKARNI, S; CHASE, A et al.Cancer research (Baltimore). 1999, Vol 59, Num 16, pp 3870-3874, issn 0008-5472Article

The genomic structure of ZNF198 and location of breakpoints in the t(8;13) myeloproliferative syndromeKULKARNI, S; REITER, A; SMEDLEY, D et al.Genomics (San Diego, Calif.). 1999, Vol 55, Num 1, pp 118-121, issn 0888-7543Article

The rate and kinetics of molecular response to donor leucocyte transfusions in chronic myeloid leukaemia patients treated for relapse after allogeneic bone marrow transplantationRAANANI, P; DAZZI, F; SOHAL, J et al.British journal of haematology. 1997, Vol 99, Num 4, pp 945-950, issn 0007-1048Article

Monitoring the efficiency of interferon-α therapy in chronic myelogenous leukemia (CML) patients by competitive polymerase chain reactionHOCHHAUS, A; LIN, F; REITER, A et al.Leukemia. 1997, Vol 11, pp 541-544, issn 0887-6924, SUP3Conference Paper

Interferon augments PML and PML/RARα expression in normal myeloid and acute promyelocytic cells and cooperates with all-trans retinoic acid to induce maturation of a retinoid-resistant promyelocytic cell lineNASON-BURCHENAL, K; GANDINI, D; BOTTO, M et al.Blood. 1996, Vol 88, Num 10, pp 3926-3936, issn 0006-4971Article

An atypical myeloproliferative disorder with t(8;13)(p11;q12): a third caseMACDONALD, D; SHEERIN, S. M; CROSS, N. C. P et al.British journal of haematology. 1994, Vol 86, Num 4, pp 879-880, issn 0007-1048Article

Null alleles of the aldolase B gene in patients with hereditary fructose intoleranceALI, M; TUNCMAN, G; CROSS, N. C. P et al.Journal of medical genetics. 1994, Vol 31, Num 6, pp 499-503, issn 0022-2593Article

No evidence for microsatellite instability or consistent loss of heterozygosity at selected loci in chronic myeloid leukaemia blast crisiSILLY, H; CHASE, A; MILLS, K. I et al.Leukemia. 1994, Vol 8, Num 11, pp 1923-1928, issn 0887-6924Conference Paper