Pascal and Francis Bibliographic Databases

Help

Search results

Your search

kw.\*:("Nav1.5")

Publication Year[py]

A-Z Z-A Frequency ↓ Frequency ↑
Export in CSV

Discipline (document) [di]

A-Z Z-A Frequency ↓ Frequency ↑
Export in CSV

Author Country

A-Z Z-A Frequency ↓ Frequency ↑
Export in CSV

Results 1 to 20 of 20

  • Page / 1
Export

Selection :

  • and

On the relationship between block of the cardiac Na+ channel and drug-induced prolongation of the QRS complexHARMER, A. R; VALENTIN, J.-P; POLLARD, C. E et al.British journal of pharmacology. 2011, Vol 164, Num 2, pp 260-273, issn 0007-1188, 14 p.Article

SAP97 and Dystrophin Macromolecular Complexes Determine Two Pools of Cardiac Sodium Channels Nav1.5 in CardiomyocytesPETITPREZ, Séverine; ZMOOS, Anne-Flore; HATEM, Stéphane N et al.Circulation research. 2011, Vol 108, Num 3, pp 294-304, issn 0009-7330, 11 p.Article

Cardiac sodium channel Nav1.5 and its associated proteinsABRIEL, H.Archives des maladies du coeur et des vaisseaux. 2007, Vol 100, Num 9, pp 787-793, issn 0003-9683, 7 p.Article

Molecular determinants of voltage-gated sodium channel regulation by the Nedd4/Nedd4-like proteinsROUGIER, Jean-Sebastien; VAN BEMMELEN, Miguel X; BRUCE, M. Christine et al.American journal of physiology. Cell physiology. 2005, Vol 57, Num 3, pp C692-C701, issn 0363-6143Article

A novel SCN5A mutation, F1344S, identified in a patient with brugada syndrome and fever-induced ventricular fibrillationKELLER, Dagmar I; HAI HUANG; JUAN ZHAO et al.Cardiovascular research. 2006, Vol 70, Num 3, pp 521-529, issn 0008-6363, 9 p.Article

Biology of cardiac sodium channel Nav1.5 expressionROOK, Martin B; EVERS, Melvin M; VOS, Marc A et al.Cardiovascular research. 2012, Vol 93, Num 1, pp 12-23, issn 0008-6363, 12 p.Article

A trafficking defective, Brugada syndrome-causing SCN5A mutation rescued by drugsVALDIVIA, Carmen R; TESTER, David J; ROK, Benjamin A et al.Cardiovascular research. 2004, Vol 62, Num 1, pp 53-62, issn 0008-6363, 10 p.Article

Mutations in sodium channel β-subunit SCN3B are associated with early-onset lone atrial fibrillationOLESEN, Morten S; JESPERSEN, Thomas; SCHMITT, Nicole et al.Cardiovascular research. 2011, Vol 89, Num 4, pp 786-793, issn 0008-6363, 8 p.Article

A novel SCN5A mutation manifests as a malignant form of long QT syndrome with perinatal onset of tachycardia/bradycardiaCHANG, Chien-Chih; ACHARFI, Said; WU, Mei-Hwan et al.Cardiovascular research. 2004, Vol 64, Num 2, pp 268-278, issn 0008-6363, 11 p.Article

A ZASP Missense Mutation, S196L, Leads to Cytoskeletal and Electrical Abnormalities in a Mouse Model of CardiomyopathyZHAOHUI LI; AI, Tomohiko; JIE CHENG et al.Circulation. Arrhythmia and electrophysiology. 2010, Vol 3, Num 6, pp 646-656, issn 1941-3149, 11 p.Article

Sodium channel mutation in irritable bowel syndrome : evidence for an ion channelopathySAITO, Yuri A; STREGE, Peter R; TESTER, David J et al.American journal of physiology. Gastrointestinal and liver physiology. 2009, Vol 59, Num 2, issn 0193-1857, G211-G218Article

Quantitation of protein kinase A-mediated trafficking of cardiac sodium channels in living cellsHALLAQ, Haifa; ZHENJIANG YANG; VISWANATHAN, Prakash C et al.Cardiovascular research. 2006, Vol 72, Num 2, pp 250-261, issn 0008-6363, 12 p.Article

Fibrillation potentials of denervated rat skeletal muscle are associated with expression of cardiac-type voltage-gated sodium channel isoform Nav1.5SEKIGUCHI, Kenji; KANDA, Fumio; MITSUI, Shigeru et al.Clinical neurophysiology. 2012, Vol 123, Num 8, pp 1650-1655, issn 1388-2457, 6 p.Article

Multiple arrhythmic syndromes in a newborn, owing to a novel mutation in SCN5ACALLOE, Kirstine; SCHMITT, Nicole; GRUBB, Soren et al.Canadian journal of physiology and pharmacology. 2011, Vol 89, Num 10, pp 723-736, issn 0008-4212, 14 p.Article

Expression of voltage-gated sodium channel α subunit in human ovarian cancerRUI GAO; YI SHEN; JING CAI et al.Oncology reports. 2010, Vol 23, Num 5, pp 1293-1299, issn 1021-335X, 7 p.Article

Molecular pharmacology of voltage-gated sodium channel expression in metastatic disease: Clinical potential of neonatal Nav1.5 in breast cancerONKAL, Rustem; DJAMGOZ, Mustafa B. A.European journal of pharmacology. 2009, Vol 625, Num 1-3, pp 206-219, issn 0014-2999, 14 p.Article

Inhibition of human Nav1.5 sodium channels by strychnine and its analogsCHUNHUA YUAN; LIRONG SUN; MENG ZHANG et al.Biochemical pharmacology. 2011, Vol 82, Num 4, pp 350-357, issn 0006-2952, 8 p.Article

Heterozygous nonsense SCN5A mutation W822X explains a simultaneous sudden infant death syndromeTURILLAZZI, Emanuela; LA ROCCA, Giampiero; ANZALONE, Rita et al.Virchows Archiv. 2008, Vol 453, Num 2, pp 209-216, issn 0945-6317, 8 p.Article

A novel C-terminal truncation SCN5A mutation from a patient with sick sinus syndrome, conduction disorder and ventricular tachycardiaTAN, Bi-Hua; ITURRALDE-TORRES, Pedro; MEDEIROS-DOMINGO, Argelia et al.Cardiovascular research. 2007, Vol 76, Num 3, pp 409-417, issn 0008-6363, 9 p.Article

TGF-β1-Mediated Fibrosis and Ion Channel Remodeling Are Key Mechanisms in Producing the Sinus Node Dysfunction Associated With SCN5A Deficiency and AgingXIAOJIN HAO; YANMIN ZHANG; HENGGUI ZHANG et al.Circulation. Arrhythmia and electrophysiology. 2011, Vol 4, Num 3, pp 397-406, issn 1941-3149, 10 p.Article

  • Page / 1