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Molecular mechanisms underlying FIP1L1-PDGFRA-mediated myeloproliferationBUITENHUIS, Miranda; VERHAGEN, Liesbeth P; COOLS, Jan et al.Cancer research (Baltimore). 2007, Vol 67, Num 8, pp 3759-3766, issn 0008-5472, 8 p.Article

Hyperéosinophilie avec transcrit de fusion FIP1L1-PDGFRA = Eosinophilia with FIP1/1-PDGFRA fusionLE BARS, Hervé; PREUDHOMME, Claude; BERNARD, Marc et al.Hématologie (Montrouge). 2007, Vol 13, Num 2, pp 122-126, issn 1264-7527, 5 p.Article

High KIT and PDGFRA are associated with shorter patients survival in gastroenteropancreatic neuroendocrine tumors, but mutations are a rare eventKNÖSEL, Thomas; YUAN CHEN; ALTENDORF-HOFMANN, Annelore et al.Journal of cancer research and clinical oncology. 2012, Vol 138, Num 3, pp 397-403, issn 0171-5216, 7 p.Article

Inflammatory fibroid polyps harbour mutations in the platelet-derived growth factor receptor alpha (PDGFRA) geneSCHILDHAUS, H.-U; CAVLAR, T; BINOT, E et al.Journal of pathology. 2008, Vol 216, Num 2, pp 176-182, issn 0022-3417, 7 p.Article

Clinical features of hypereosinophilic syndrome : FIP1L1-PDGFRA fusion gene-positive disease is a distinct clinical entity with myeloproliferative features and a poor response to corticosteroidMIYAZAWA, Keisuke; KAKAZU, Naoki; OHYASHIKI, Kazuma et al.International journal of hematology. 2007, Vol 85, Num 1, pp 5-10, issn 0925-5710, 6 p.Article

FIP1L1-PDGFRA in chronic eosinophilic leukemia and BCR-ABL1 in chronic myeloid leukemia affect different leukemic cellsCRESCENZI, B; CHASE, A; JONES, A. V et al.Leukemia. 2007, Vol 21, Num 3, pp 397-402, issn 0887-6924, 6 p.Article

PDGFRA Immunostaining Can Help in the Diagnosis of Gastrointestinal Stromal TumorsMISELLI, Francesca; MILLEFANTI, Clara; CONCA, Elena et al.The American journal of surgical pathology. 2008, Vol 32, Num 5, pp 738-743, issn 0147-5185, 6 p.Article

The severity of FlP1L1 -PDGFRA -positive chronic eosinophilic leukaemia is associated with polymorphic variation at the IL5RA locusBURGSTALLER, S; KREIL, S; WALZ, C et al.Leukemia. 2007, Vol 21, Num 12, pp 2428-2432, issn 0887-6924, 5 p.Article

KIT and PDGFRa mutational analyses of mixed cell-type gastrointestinal stromal tumoursWONG, N. A. C. S; MANGWANA, S.Histopathology. 2007, Vol 51, Num 6, pp 758-762, issn 0309-0167, 5 p.Article

Plexiform Fibromyxoma: A Distinctive Benign Gastric Antral Neoplasm Not to be Confused With a Myxoid GISTMIETTINEN, Markku; MAKHLOUF, Hala R; SOBIN, Leslie H et al.The American journal of surgical pathology. 2009, Vol 33, Num 11, pp 1624-1632, issn 0147-5185, 9 p.Article

T-cell abnormalities are present at high frequencies in patients with hypereosinophilic syndromeHEIBIG, Grzegorz; WIECZORKIEWICZ, Agata; DZIACZKOWSKA-SUSZEK, Joanna et al.Haematologica (Roma). 2009, Vol 94, Num 9, pp 1236-1241, issn 0390-6078, 6 p.Article

Mechanism for the differentiation of EoL-1 cells into eosinophils by histone deacetylase inhibitorsKANEKO, Motoko; ISHIHARA, Kenji; TAKAHASHI, Aki et al.International archives of allergy and immunology. 2007, Vol 143, pp 28-32, issn 1018-2438, 5 p., SUP1Conference Paper

Expression, mutation and copy number analysis of platelet-derived growth factor receptor A (PDGFRA) and its ligand PDGFA in gliomasMARTINHO, O; LONGATTO, A; TAMBER, N et al.British journal of cancer. 2009, Vol 101, Num 6, pp 973-982, issn 0007-0920, 10 p.Article

Hypereosinophilic syndrome: A multicenter, retrospective analysis of clinical characteristics and response to therapyOGBOGU, Princess U; BOCHNER, Bruce S; ROTHENBERG, Marc E et al.Journal of allergy and clinical immunology. 2009, Vol 124, Num 6, pp 1319-1325, issn 0091-6749, 7 p.Article

Practical approach to the patient with hypereosinophiliaROUFOSSE, Florence; WELLER, Peter F.Journal of allergy and clinical immunology. 2010, Vol 126, Num 1, pp 39-44, issn 0091-6749, 6 p.Article

Prognostic Relevance of KIT and PDGFRA Mutations in Gastrointestinal Stromal TumorsBRAGGIO, Esteban; DE ALMEIDA BRAGGIO, Danielle; RENAULT, Ilana Zalcberg et al.Anticancer research. 2010, Vol 30, Num 6, pp 2407-2414, issn 0250-7005, 8 p.Article

KIT and PDGFRα mutations in 104 patients with gastrointestinal stromal tumors (GISTs) : a population-based studyBRACONI, C; BRACCI, R; ROSSI, G et al.Annals of oncology. 2008, Vol 19, Num 4, pp 706-710, issn 0923-7534, 5 p.Article

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Differentially expressed proteins in gastrointestinal stromal tumors with KIT and PDGFRA mutationsHYUN JU KANG; KWI HYE KOH; EUNGI YANG et al.Proteomics (Weinheim. Print). 2006, Vol 6, Num 4, pp 1151-1157, issn 1615-9853, 7 p.Article

Gastrointestinal stromal tumors (GISTs) with KIT and PDGFRA mutations have distinct gene expression profilesSUBRAMANIAN, Subbaya; WEST, Robert B; MONTGOMERY, Kelli et al.Oncogene (Basingstoke). 2004, Vol 23, Num 47, pp 7780-7790, issn 0950-9232, 11 p.Article

Cell clonality in hypereosinophilic syndrome : what pathogenetic role?GALIMBERTI, S; CIABATTI, E; OTTIMO, F et al.Clinical and experimental rheumatology (Testo stampato). 2007, Vol 25, Num 1, pp 17-22, issn 0392-856X, 6 p.Article

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A combination of cytomorphology, cytogenetic analysis, fluorescence in situ hybridization and reverse transcriptase polymerase chain reaction for establishing clonality in cases of persisting hypereosinophiliaBACHER, Ulrike; REITER, Andreas; HAFERLACH, Torsten et al.Haematologica (Roma). 2006, Vol 91, Num 6, pp 817-820, issn 0390-6078, 4 p.Article

Comparison of morphological, immunohistochemical, and molecular genetic features of inflammatory fibroid polyps (Vaneks tumors)DAUM, Ondrej; HATLOVA, Jana; MANDYS, Vaclav et al.Virchows Archiv. 2010, Vol 456, Num 5, pp 491-497, issn 0945-6317, 7 p.Article