EVI1 is critical for the pathogenesis of a subset of MLL-AF9-rearranged AMLsBINDELS, Eric M. J; HAVERMANS, Marije; BERNA BEVERLOO, H et al.Blood. 2012, Vol 119, Num 24, pp 5838-5849, issn 0006-4971, 12 p.Article

An insertional mutagenesis screen identifies genes that cooperate with Mll-AF9 in a murine leukemogenesis modelBERGERSON, Rachel J; COLLIER, Lara S; DANHUA FAN et al.Blood. 2012, Vol 119, Num 19, pp 4512-4523, issn 0006-4971, 12 p.Article

High BRE expression predicts favorable outcome in adult acute myeloid leukemia, in particular among MLL-AF9-positive patientsNOORDERMEER, Sylvie M; SANDERS, Mathijs A; GILISSEN, Christian et al.Blood. 2011, Vol 118, Num 20, pp 5613-5621, issn 0006-4971, 9 p.Article

Transformation from committed progenitor to leukaemia stem cell initiated by MLL-AF9KRIVTSOV, Andrei V; TWOMEY, David; GOLUB, Todd R et al.Nature (London). 2006, Vol 442, Num 7104, pp 818-822, issn 0028-0836, 5 p.Article

Apoptotic stimuli initiate MLL-AF9 translocations that are transcribed in cells capable of divisionBETTI, Christopher J; VILLALOBOS, Michael J; DIAZ, Manuel O et al.Cancer research (Baltimore). 2003, Vol 63, Num 6, pp 1377-1381, issn 0008-5472, 5 p.Article

DNA structural properties of AF9 are similar to MLL and could act as recombination hot spots resulting in MLL/AF9 translocations and leukemogenesisSTRISSEL, P. L; STRICK, R; TOMEK, R. J et al.Human molecular genetics (Print). 2000, Vol 9, Num 11, pp 1671-1679, issn 0964-6906Article

A murine Mll-AF4 knock-in model results in lymphoid and myeloid deregulation and hematologic malignancy. CommentaryGREAVES, Mel; SO, C. W. Eric; WEILI CHEN et al.Blood. 2006, Vol 108, Num 2, issn 0006-4971, 416-417,669-677 [11 p.]Article

Targeted down-regulation of MLL-AF9 with antisense oligodeoxyribonucleotide reduces the expression of the HOXA7 and -A10 genes and induces apoptosis in a human leukemia cell line, THP-1KAWAGOE, H; KAWAGOE, R; SANO, K et al.Leukemia. 2001, Vol 15, Num 11, pp 1743-1749, issn 0887-6924Article

The partial nontandem duplication of the MLL (ALL1) gene is a novel rearrangement that generates three distinct fusion transcripts in B-cell acute lymphoblastic leukemiaWHITMAN, Susan P; STROUT, Matthew P; CALIGIURI, Michael A et al.Cancer research (Baltimore). 2001, Vol 61, Num 1, pp 59-63, issn 0008-5472Article

Genes on chromosomes 4, 9, and 19 involved in 11q23 abnormalities in acute leukemia share sequence homology and/or common motifsNAKAMURA, T; ALDER, H; CROCE, C. M et al.Proceedings of the National Academy of Sciences of the United States of America. 1993, Vol 90, Num 10, pp 4631-4635, issn 0027-8424Article

MLL-AF9 and FLT3 cooperation in acute myelogenous leukemia : development of a model for rapid therapeutic assessmentSTUBBS, M. C; KIM, Y. M; KRIVTSOV, A. V et al.Leukemia. 2008, Vol 22, Num 1, pp 66-77, issn 0887-6924, 12 p.Article

Two acute monocytic leukemia (AML-M5a) cell lines (MOLM-13 and MOLM-14) with interclonal phenotypic heterogeneity showing MLL-AF9 fusion resulting from an occult chromosome insertion, ins(11;9)(q23;p22p23)MATSUO, Y; MACLEOD, R. A. F; ORITA, K et al.Leukemia. 1997, Vol 11, Num 9, pp 1469-1477, issn 0887-6924Article