au.\*:("MARSHALL, Connie A")
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Decreased Fetal Size Is Associated With p-Cell Hyperfunction in Early Life and Failure With Age. CommentaryLEAHY, Jack L; CHAKRAVARTHY, Manu V; YIMIN ZHU et al.Diabetes (New York, NY). 2008, Vol 57, Num 10, issn 0012-1797, 2563-2564, 2698-2707 [12 p.]Article
Signaling elements involved in the metabolic regulation of mTOR by nutrients, incretins, and growth factors in isletsKWON, Guim; MARSHALL, Connie A; PAPPAN, Kirk L et al.Diabetes (New York, NY). 2004, Vol 53, pp S225-S232, issn 0012-1797, SUP3Conference Paper
Metabolic and autocrine regulation of the mammalian target of rapamycin by pancreatic β-cellsMCDANIEL, Michael L; MARSHALL, Connie A; PAPPAN, Kirk L et al.Diabetes (New York, NY). 2002, Vol 51, Num 10, pp 2877-2885, issn 0012-1797, 9 p.Article
Glycogen Synthase Kinase-3 and Mammalian Target of Rapamycin Pathways Contribute to DNA Synthesis, Cell Cycle Progression, and Proliferation in Human IsletsHUI LIU; REMEDI, Maria S; PAPPAN, Kirk L et al.Diabetes (New York, NY). 2009, Vol 58, Num 3, pp 663-672, issn 0012-1797, 10 p.Article
Metabolic regulation by leucine of translation initiation through the mTOR-signaling pathway by pancreatic β-cellsGUANG XU; GUIM KWON; CRUZ, Wilhelm S et al.Diabetes (New York, NY). 2001, Vol 50, Num 2, pp 353-360, issn 0012-1797Article