The roles of intracellular protein-degradation pathways in neurodegeneration : Neurodegeneration
- Auteur
- RUBINSZTEIN, David C1
[1] Department of Medical Genetics, Cambridge Institute for Medical Research, Addenbrooke's Hospital, Hills Road, Cambridge CB2 2XY, United Kingdom - Source
Nature (London). 2006, Vol 443, Num 7113, pp 780-786, 7 p ; ref : 76 ref
- CODEN
- NATUAS
- ISSN
- 0028-0836
- Domaine scientifique
- Multidisciplinary
- Editeur
- Nature Publishing, London
- Pays de publication
- United Kingdom
- Type de document
- Article
- Langue
- English
- Mot clé (fr)
- Agrégation Chorée Huntington Homme Parkinson maladie Pathogénie Protéine Ubiquitine Encéphale pathologie Extrapyramidal syndrome Maladie dégénérative Maladie héréditaire Système nerveux central pathologie Système nerveux pathologie
- Mot clé (en)
- Aggregation Huntington disease Human Parkinson disease Pathogenesis Protein Ubiquitin Cerebral disorder Extrapyramidal syndrome Degenerative disease Genetic disease Central nervous system disease Nervous system diseases
- Mot clé (es)
- Agregación Corea Huntington Hombre Parkinson enfermedad Patogenia Proteína Ubiquitina Encéfalo patología Extrapiramidal síndrome Enfermedad degenerativa Enfermedad hereditaria Sistema nervosio central patología Sistema nervioso patología
- Classification
- Pascal
- 002 Sciences biologiques et medicales / 002B Sciences medicales / 002B17 Neurologie / 002B17G Maladies dégénératives et hérédodégénératives du système nerveux. Leucodystrophies. Maladies à prions
- Discipline
- Neurology
- Provenance
- Inist-CNRS
- Base de données
- PASCAL
- Identifiant INIST
- 18203032
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/nature05291 
https://dx.doi.org/10.1038/nature05291
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