Dexamethasone reduces bilirubin-induced toxicity and IL-8 and MCP-1 release in human NT2-N neurons
- Auteur
- ALMAAS, Runar1 2 ; HANKØ, Erik1 2 ; EIRIK MOLLNES, Tom3 4 ; ROOTWELT, Terje2 4
[1] Department of Pediatric Research, Oslo University Hospital Rikshospitalet, 0027 Oslo, Norway
[2] Department of Pediatrics, Oslo University Hospital, Rikshospitalet, 0027 Oslo, Norway
[3] Institute of Immunology, University of Oslo and Oslo University Hospital Rikshospitalet, 0027 Oslo, Norway
[4] University of Oslo, Oslo, Norway - Source
Brain research. 2012, Vol 1458, pp 12-17, 6 p ; ref : 3/4 p
- CODEN
- BRREAP
- ISSN
- 0006-8993
- Domaine scientifique
- Cognition; Neurology; Physiology, morphology
- Editeur
- Elsevier, Amsterdam
- Pays de publication
- Netherlands
- Type de document
- Article
- Langue
- English
- Mot clé auteur
- Bilirubin CXCL-10 Cell death Cytokine Dexamethasone IL-8 IP-10 MCP-1 NT2-N neuron
- Mot clé (fr)
- Bilirubine Chimiokine CXC Cytokine Dexaméthasone Homme Hyperbilirubinémie In vitro Interleukine 8 Libération Maladie métabolique Mort cellulaire Neurone Protéine MCP1 Toxicité
- Mot clé (en)
- Bilirubin CXC chemokine Cytokine Dexamethasone Human Hyperbilirubinemia In vitro Interleukin 8 Release Metabolic diseases Cell death Neuron Monocyte chemoattractant protein 1 Toxicity
- Mot clé (es)
- Bilirrubina Quimioquina CXC Citoquina Dexametasona Hombre Hiperbilirrubinemia In vitro Interleuquina 8 Liberación Metabolismo patología Muerte celular Neurona Proteina quimiocaptante monocitos 1 Toxicidad
- Classification
- Pascal
- 002 Sciences biologiques et medicales / 002B Sciences medicales / 002B22 Maladies metaboliques / 002B22E Autres maladies métaboliques / 002B22E02 Pigments (porphyries, hyperbilirubinémies...)
- Discipline
- Metabolic diseases
- Provenance
- Inist-CNRS
- Base de données
- PASCAL
- Identifiant INIST
- 25928757
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1016/j.brainres.2012.04.007 
https://dx.doi.org/10.1016/j.brainres.2012.04.007
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